EVOLUTIONIST CLAIMS &THE FACTS
In previous chapters, we examined the invalidity of the theory
of evolution in terms of the bodies of evidence found in fossils
and from the standpoint of molecular biology. In this chapter, we
will address a number of biological phenomena and concepts presented
as theoretical evidence by evolutionists. These topics are particularly
important for they show that there is no scientific finding that
supports evolution and instead reveal the extent of the distortion
and hoodwink employed by evolutionists.
Variations and Species
Variation, a term used in genetics, refers to a genetic event that
causes the individuals or groups of a certain type or species to
possess different characteristics from one another. For example,
all the people on earth carry basically the same genetic information,
yet some have slanted eyes, some have red hair, some have long noses,
and others are short of stature, all depending on the extent of
the variation potential of this genetic information.
| 
DID
WHALES EVOLVE FROM BEARS?
In The Origin of Species, Darwin asserted that whales had
evolved from bears that tried to swim! Darwin mistakenly supposed
that the possibilities of variation within a species were
unlimited. 20th century science has shown this evolutionary
scenario to be imaginary.
|
Evolutionists predicate the variations within a species as evidence
to the theory. However, variation does not constitute
evidence for evolution because variations are but the outcomes of
different combinations of already existing genetic information and
they do not add any new characteristic to the genetic information.
The important thing for the theory of evolution, however, is the question
of how brand-new information to make a brand-new species could come
about. Variation always takes place within the limits of genetic
information. In the science of genetics, this limit is called the
"gene pool". All of the characteristics present in the gene pool
of a species may come to light in various ways due to variation.
For example, as a result of variation, varieties that have relatively
longer tails or shorter legs may appear in a certain species of
reptile, since information for both long-legged and short-legged
forms may exist in the gene pool that species. However, variations
do not transform reptiles into birds by adding wings or feathers
to them, or by changing their metabolism. Such a change requires
an increase in the genetic information of the living thing, which
is certainly not possible through variations.
Darwin was not aware of this fact when he formulated
his theory. He thought that there was no limit to variations. In
an article he wrote in 1844 he stated: "That
a limit to variation does exist in nature is assumed by most authors,
though I am unable to discover a single fact on which this belief
is grounded".151 In The Origin of Species
he cited different examples of variations as the most important
evidence for his theory.
For instance, according to Darwin, animal breeders who mated different
varieties of cattle in order to bring about new varieties that produced
more milk, were ultimately going to transform them into a different
species. Darwin's notion of "unlimited variation" is best seen in
the following sentence from The Origin of Species:
I can see no difficulty in a race of bears
being rendered, by natural selection, more and more aquatic in
their habits, with larger and larger mouths, till a creature was
produced as monstrous as a whale.152
The reason Darwin cited such a far-fetched example was the primitive
understanding of science in his day. Since then, in the 20th century,
science has posited the principle of "genetic
stability" (genetic homeostasis), based on the results of
experiments conducted on living things. This principle holds that,
since all mating attempts carried out to produce new variations
have been inconclusive, there are strict barriers
among different species of living things. This meant that
it was absolutely impossible for animal breeders to convert cattle
into a different species by mating different variations of them,
as Darwin had postulated.
Norman Macbeth, who disproved Darwinism in his book Darwin Retried,
states:
The heart of the problem is whether living
things do indeed vary to an unlimited extent... The
species look stable. We have all heard of disappointed
breeders who carried their work to a certain point only to see
the animals or plants revert to where they had started. Despite
strenuous efforts for two or three centuries, it has never been
possible to produce a blue rose or a black tulip.153
Luther Burbank, considered the most competent
breeder of all time, expressed this fact when he said, "there are
limits to the development possible, and these limits follow a law."
154 The Danish scientist W. L. Johannsen sums
the matter up this way:
The variations upon which Darwin and Wallace
had placed their emphasis cannot be selectively pushed beyond
a certain point, that such a variability
does not contain the secret of 'indefinite departure.155
In the same way, the different finches that Darwin saw on the
Galapagos Islands are another example of variation that is no
evidence for "evolution". Recent observations have
revealed that the finches did not undergo an unlimited variation
as Darwin's theory presupposed. Moreover, most of the different
types of finches which Darwin thought represented 14 distinct
species actually mated with one another, which means that they
were variations that belonged to the same species. Scientific
observation shows that the finch beaks, which have been mythicized
in almost all evolutionist sources, are in fact an example of "variation";
therefore, they do not constitute evidence for the theory of
evolution. For example, Peter and Rosemary Grant, who spent years
observing the finch varieties in the Galapagos Islands looking
for evidence for Darwinistic evolution, were forced to conclude
that no "evolution" that leads to the emergence of
new traits ever takes place there.
Antibiotic Resistance and DDT Immunity are not Evidence for Evolution
One of the biological concepts that evolutionists try to present
as evidence for their theory is the resistance of bacteria to antibiotics.
Many evolutionist sources show antibiotic resistance as "an example
of the development of living things by advantageous mutations".
A similar claim is also made for the insects which build immunity
to insecticides such as DDT.
However, evolutionists are mistaken on this subject too.
Antibiotics are "killer molecules" that are produced by micro-organisms
to fight other micro-organisms. The first antibiotic was penicillin,
discovered by Alexander Fleming in 1928. Fleming realized that mould
produced a molecule that killed the Staphylococcus bacterium, and
this discovery marked a turning point in the world of medicine.
Antibiotics derived from micro-organisms were used against bacteria
and the results were successful.
Soon, something new was discovered. Bacteria build immunity to
antibiotics over time. The mechanism works like this: A large proportion
of the bacteria that are subjected to antibiotics die, but some
others, which are not affected by that antibiotic, replicate rapidly
and soon make up the whole population. Thus, the entire population
becomes immune to antibiotics.
Evolutionists try to present this as "the evolution of bacteria
by adapting to conditions".
The truth, however, is very different from this superficial interpretation.
One of the scientists who has done the most detailed research into
this subject is the Israeli biophysicist Lee Spetner, who is also
known for his book Not by Chance published in 1997. Spetner maintains
that the immunity of bacteria comes about by two different mechanisms,
but neither of them constitutes evidence for the theory of evolution.
These two mechanisms are:
1) The transfer of resistance genes already extant in bacteria.
2) The building of resistance as a result of losing genetic data
because of mutation.
Professor Spetner explains the first mechanism in an article published
in 2001:
Some microorganisms are endowed with genes that
grant resistance to these antibiotics. This resistance can take
the form of degrading the antibiotic molecule or of ejecting it
from the cell... The organisms having these genes can transfer them
to other bacteria making them resistant as well. Although the resistance
mechanisms are specific to a particular antibiotic, most pathogenic
bacteria have... succeeded in accumulating several sets of genes
granting them resistance to a variety of antibiotics.156
Spetner then goes on to say that this is not "evidence for evolution":
The acquisition of antibiotic resistance in
this manner... is not the kind that can serve as a prototype for
the mutations needed to account for Evolution. The genetic changes
that could illustrate the theory must not only add information
to the bacterium's genome, they must add new information to the
biocosm. The horizontal transfer of genes only spreads around
genes that are already in some species.157
So, we cannot talk of any evolution here, because no new genetic
information is produced: genetic information that already exists
is simply transferred between bacteria.
The second type of immunity, which comes about as a result of mutation,
is not an example of evolution either. Spetner writes:
...A microorganism can sometimes acquire resistance
to an antibiotic through a random substitution of a single nucleotide...
Streptomycin, which was discovered by Selman Waksman and Albert
Schatz and first reported in 1944, is an antibiotic against which
bacteria can acquire resistance in this way. But although the
mutation they undergo in the process is beneficial to the microorganism
in the presence of streptomycin, it cannot serve as a prototype
for the kind of mutations needed by NDT[Neo Darwinian Theory].
The type of mutation that grants resistance to streptomycin is
manifest in the ribosome and degrades its molecular match with
the antibiotic molecule. This change in the surface of the microorganism's
ribosome prevents the streptomycin molecule from attaching and
carrying out its antibiotic function. It turns out that this degradation
is a loss of specificity and therefore a loss of information.
The main point is that (Evolution) cannot be achieved by mutations
of this sort, no matter how many of them there are. Evolution
cannot be built by accumulating mutations that only degrade specificity.158
To sum up, a mutation impinging on a bacterium's ribosome makes
that bacterium resistant to streptomycin. The reason for this is
the "decomposition" of the ribosome by mutation. That is, no new
genetic information is added to the bacterium. On the contrary,
the structure of the ribosome is decomposed, that is to say, the
bacterium becomes "disabled". (Also, it has been discovered that
the ribosome of the mutated bacterium is less functional than that
of normal bacterium). Since this "disability" prevents the antibiotic
from attaching onto the ribosome, "antibiotic resistance" develops.
Finally, there is no example of mutation that
"develops the genetic information".
The same situation holds true for the immunity that insects develop
to DDT and similar insecticides. In most of these instances, immunity
genes that already exist are used. The evolutionist biologist Francisco
Ayala admits this fact, saying, "The genetic variants required for
resistance to the most diverse kinds of pesticides were apparently
present in every one of the populations exposed to these man-made
compounds."159 Some other
examples explained by mutation, just as with the ribosome mutation
mentioned above, are phenomena that cause "genetic information deficit"
in insects.
In this case, it cannot be claimed that the immunity mechanisms
in bacteria and insects constitute evidence for the theory of evolution.
That is because the theory of evolution is based on the assertion
that living things develop through mutations. However, Spetner explains
that neither antibiotic immunity nor any other biological phenomena
indicate such an example of mutation:
The mutations needed for macroevolution have
never been observed. No random mutations that could represent the
mutations required by Neo-Darwinian Theory that have been examined
on the molecular level have added any information. The question
I address is: Are the mutations that have been observed the kind
the theory needs for support? The answer turns out to be NO!160
The Fallacy of Vestigial Organs
For a long time, the concept of "vestigial organs" appeared frequently
in evolutionist literature as "evidence" of evolution. Eventually,
it was silently put to rest when this was proved to be invalid.
But some evolutionists still believe in it, and from time to time
someone will try to advance "vestigial organs" as important evidence
of evolution.
The notion of "vestigial organs" was first put forward a century
ago. As evolutionists would have it, there existed in the bodies
of some creatures a number of non-functional organs. These had been
inherited from progenitors and had gradually become vestigial from
lack of use.
| 
All instances of vestigial organs
have been disproved in time. For example the semicircular
fold in the eye, which was mentioned in the Origins as a vestigial
structure, has been shown to be fully functional in our time,
though its function was unknown in Darwin's time. This organ
lubricates the eyeball.
|
The whole assumption is quite unscientific, and is based entirely
on insufficient knowledge. These "non-functional
organs" were in fact organs whose "functions
had not yet been discovered". The best indication of this
was the gradual yet substantial decrease in evolutionists' long
list of vestigial organs. S.R. Scadding, an evolutionist himself,
concurred with this fact in his article "Can vestigial organs constitute
evidence for evolution?" published in the journal Evolutionary Theory:
Since it is not possible to unambiguously identify
useless structures, and since the structure of the argument used
is not scientifically valid, I conclude that
"vestigial organs" provide no special evidence for the theory of
evolution.161
The list of vestigial organs that was made by
the German Anatomist R. Wiedersheim in 1895 included approximately
100 organs, including the appendix and coccyx. As science progressed,
it was discovered that all of the organs in Wiedersheim's list in
fact had very important functions. For instance, it was discovered
that the appendix, which was supposed to be a "vestigial organ",
was in fact a lymphoid organ that fought against infections in the
body. This fact was made clear in 1997: "Other bodily organs and
tissues-the thymus, liver, spleen, appendix,
bone marrow, and small collections of lymphatic tissue such
as the tonsils in the throat and Peyer's patch in the small intestine-are
also part of the lymphatic system. They too help
the body fight infection."162
It was also discovered that the tonsils, which were included in
the same list of vestigial organs, had a significant role in protecting
the throat against infections, particularly until adolescence. It
was found that the coccyx at the lower end of the vertebral column
supports the bones around the pelvis and is the convergence point
of some small muscles and for this reason, it would not be possible
to sit comfortably without a coccyx. In the years that followed,
it was realised that the thymus triggered
the immune system in the human body by activating the T cells, that
the pineal gland was in charge of the
secretion of some important hormones, that the thyroid gland was
effective in providing steady growth in babies and children, and
that the pituitary gland controlled the correct functioning of many
hormone glands. All of these were once conside-red to be "vestigial
organs". Finally, the semi-lunar fold in the eye, which was
referred to as a vestigial organ by Darwin, has been found in fact
to be in charge of cleansing and lubricating the eyeball.
There was a very important logical error in
the evolutionist claim regarding vestigial organs. As we have just
seen, this claim was that the vestigial organs in living things
were inherited from their ancestors. However, some of the alleged
"vestigial" organs are not found in the species alleged to be the
ancestors of human beings! For example, the appendix does not exist
in some ape species that are said to be ancestors of man. The famous
biologist H. Enoch, who challenged the theory of vestigial organs,
expressed this logical error as follows:
Apes possess an appendix, whereas their less immediate relatives,
the lower apes, do not; but it appears again among the still lower
mammals such as the opossum. How can the evolutionists account
for this?163
Simply put, the scenario of vestigial organs put forward by evolutionists
contains a number of serious logical flaws, and has in any case
been proven to be scientifically untrue. There exists not one inherited
vestigial organ in the human body, since human beings did not evolve
from other creatures as a result of chance, but were created in
their current, complete, and perfect form.
  
151
Loren C. Eiseley, The Immense Journey, Vintage Books, 1958, p. 186.
152 Charles Darwin, The Origin of Species: A Facsimile
of the First Edition, Harvard University Press, 1964, p. 184.
153 Norman Macbeth, Darwin Retried: An Appeal
to Reason, Harvard Common Press, New York: 1971, p. 33.
154 Ibid, p. 36.
155 Loren Eiseley, The Immense Journey, Vintage
Books, 1958. p. 227.
156 Dr. Lee Spetner, "Lee Spetner/Edward
Max Dialogue: Continuing an exchange with Dr. Edward E. Max",
2001, http://www.trueorigin.org/spetner2.asp.
157 Ibid.
158 Ibid.
159 Francisco J. Ayala, "The Mechanisms of Evolution",
Scientific American, Vol 239, September 1978, p. 64.
160 Dr. Lee Spetner, "Lee Spetner/Edward
Max Dialogue: Continuing an exchange with Dr. Edward E. Max",
2001, http://www.trueorigin.org/spetner2.asp.
161 S. R. Scadding, "Do 'Vestigial Organs' Provide
Evidence for Evolution?", Evolutionary Theory, Vol 5, May 1981,
p. 173.
162 The Merck Manual of Medical Information, Home
edition, New Jersey: Merck & Co., Inc. The Merck Publishing
Group, Rahway, 1997
163 H. Enoch, Creation and Evolution, New York:
1966, p. 18-19.
|